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dc.contributor.authorEzeala, C.C.
dc.contributor.authorChukwurah, E.
dc.identifier.citationEzeala,C.C. and Chukwurah, E. (1994) Hypothyroxinemia in Acquired Immune Deficiency Syndrome (AIDS), The Central African Journal of Medicine,(CAJM), vol. 40, no.8, pp.226-229. Harare: CAJM.en
dc.descriptionA journal article on HIV/Aids.en
dc.description.abstractAcquired immune deficiency syndrome (AIDS), a disease predictive of a defect in the cell-mediated immunity, is caused by the human immuno-deficiency virus (HIV).1'1 Infection with this virus results in a gradual but progressive deterioration of immune function leading to disease manifestation. The syndrome is characterised by generalised infection and lymphad- enopathy, and is often associated with multisystem disorders. Patients with AIDS have been reported to have a high frequency of endocrine abnormalities, mostly affecting the adrenal gland, but also the thyroid and other endocrine organs.4-8 These lesions may result from necrosis, infection of the organs, or malignant transformations, presenting as Kaposi sarcomasen
dc.publisherFaculty of Medicine, Central African Journal of Medicine (CAJM), University of Zimbabwe (UZ)en
dc.titleHypothyroxinemia in Acquired Immune Deficiency Syndrome (AIDS)en
dc.rights.holderUniversity of Zimbabwe (UZ)en

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